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For example, 15 mg of natural alpha-tocopherol would equal The corresponding value for synthetic alpha-tocopherol would be Numerous foods provide vitamin E. Nuts, seeds, and vegetable oils are among the best sources of alpha-tocopherol, and significant amounts are available in green leafy vegetables and fortified cereals see Table 2 for a more detailed list [ 9 ].

Most vitamin E in American diets is in the form of gamma-tocopherol from soybean, canola, corn, and other vegetable oils and food products [ 4 ]. FDA developed DVs to help consumers compare the nutrient contents of foods and dietary supplements within the context of a total diet.

The DV for vitamin E is 15 mg for adults and children age 4 years and older [ 7 ]. FDA does not require food labels to list vitamin E content unless vitamin E has been added to the food.

Department of Agriculture’s USDA’s FoodData Central website lists the nutrient content of many foods, including, in some cases, the amounts of alpha-, beta-, gamma-, and delta-tocopherol.

The USDA also provides a comprehensive list of foods containing vitamin E arranged by nutrient content and by food name. Supplements of vitamin E typically provide only alpha-tocopherol, although “mixed” products containing other tocopherols and even tocotrienols are available.

Naturally occurring alpha-tocopherol exists in one stereoisomeric form. In contrast, synthetically produced alpha-tocopherol contains equal amounts of its eight possible stereoisomers; serum and tissues maintain only four of these stereoisomers [ 6 ]. A given amount of synthetic alpha-tocopherol all rac -alpha-tocopherol; commonly labeled as “DL” or “dl” is therefore only half as active as the same amount by weight in mg of the natural form RRR -alpha-tocopherol; commonly labeled as “D” or “d”.

These amounts are substantially higher than the RDAs. Alpha-tocopherol in dietary supplements and fortified foods is often esterified to prolong its shelf life while protecting its antioxidant properties.

The body hydrolyzes and absorbs these esters alpha-tocopheryl acetate and succinate as efficiently as alpha-tocopherol [ 6 ]. These intake estimates might be low, however, because the amounts and types of fat added during cooking are often unknown and not accounted for [ 6 ]. The FNB suggests that mean intakes of vitamin E among healthy adults are probably higher than the RDA but cautions that low-fat diets might provide insufficient amounts unless people make their food choices carefully by, for example, increasing their intakes of nuts, seeds, fruits, and vegetables [ 6 , 10 ].

Frank vitamin E deficiency is rare and overt deficiency symptoms have not been found in healthy people who obtain little vitamin E from their diets [ 6 ]. Vitamin E supplementation in these infants might reduce the risk of some complications, such as those affecting the retina, but they can also increase the risk of infections [ 13 ]. Because the digestive tract requires fat to absorb vitamin E, people with fat-malabsorption disorders are more likely to become deficient than people without such disorders.

Deficiency symptoms include peripheral neuropathy, ataxia, skeletal myopathy, retinopathy, and impairment of the immune response [ 6 , 14 ]. People with Crohn’s disease, cystic fibrosis, or an inability to secrete bile from the liver into the digestive tract, for example, often pass greasy stools or have chronic diarrhea; as a result, they sometimes require water-soluble forms of vitamin E, such as tocopheryl polyethylene glycol succinate [ 1 ].

Vitamin E deficiency secondary to abetalipoproteinemia causes such problems as poor transmission of nerve impulses, muscle weakness, and retinal degeneration that leads to blindness [ 15 ]. Ataxia and vitamin E deficiency AVED is another rare, inherited disorder in which the liver’s alpha-tocopherol transfer protein is defective or absent.

People with AVED have such severe vitamin E deficiency that they develop nerve damage and lose the ability to walk unless they take large doses of supplemental vitamin E [ 16 ]. Many claims have been made about vitamin E’s potential to promote health and prevent and treat disease.

The mechanisms by which vitamin E might provide this protection include its function as an antioxidant and its roles in anti-inflammatory processes, inhibition of platelet aggregation, and immune enhancement. A primary barrier to characterizing the roles of vitamin E in health is the lack of validated biomarkers for vitamin E intake and status to help relate intakes to valid predictors of clinical outcomes [ 6 ].

This section focuses on four diseases and disorders in which vitamin E might be involved: heart disease, cancer, eye disorders, and cognitive decline. Evidence that vitamin E could help prevent or delay coronary heart disease CHD comes from several sources. In vitro studies have found that the nutrient inhibits oxidation of low-density lipoprotein LDL cholesterol, thought to be a crucial initiating step for atherosclerosis [ 6 ].

Vitamin E might also help prevent the formation of blood clots that could lead to a heart attack or venous thromboembolism [ 17 ]. Several observational studies have associated lower rates of heart disease with higher vitamin E intakes. Among a group of 5, Finnish men and women followed for a mean of 14 years, higher vitamin E intakes from food were associated with decreased mortality from CHD [ 19 ].

However, randomized clinical trials cast doubt on the efficacy of vitamin E supplements to prevent CHD [ 20 ]. HOPE-TOO found that vitamin E provided no significant protection against heart attacks, strokes, unstable angina, or deaths from cardiovascular disease or other causes after 7 years of treatment. Not only did the supplements provide no cardiovascular benefits, but all-cause mortality was significantly higher in the women taking the supplements. The investigators found no significant differences in rates of overall cardiovascular events combined nonfatal heart attacks, strokes, and cardiovascular deaths or all-cause mortality between the groups.

Furthermore, use of vitamin E was associated with a significantly increased risk of hemorrhagic stroke. In general, clinical trials have not provided evidence that routine use of vitamin E supplements prevents cardiovascular disease or reduces its morbidity and mortality.

However, participants in these studies have been largely middle-aged or elderly individuals with demonstrated heart disease or risk factors for heart disease.

Some researchers have suggested that understanding the potential utility of vitamin E in preventing CHD might require longer studies in younger participants taking higher doses of the supplement [ 27 ]. Further research is needed to determine whether supplemental vitamin E has any protective value for younger, healthier people at no obvious risk of CHD.

Antioxidant nutrients like vitamin E protect cell constituents from the damaging effects of free radicals that, if unchecked, might contribute to cancer development [ 9 ].

Vitamin E might also block the formation of carcinogenic nitrosamines formed in the stomach from nitrites in foods and protect against cancer by enhancing immune function [ 28 ]. Unfortunately, human trials and surveys that have attempted to associate vitamin E intake with cancer incidence have found that vitamin E is not beneficial in most cases.

Based in part on the promising results of this study, a large randomized clinical trial, called the SELECT trial, began in to determine whether 7—12 years of daily supplementation with IU of synthetic vitamin E mg, as dl -alpha-tocopheryl acetate , with or without selenium mcg, as L-selenomethionine , reduced the number of new prostate cancers in 35, healthy men age 50 and older. The trial was discontinued in October when an analysis found that the supplements, taken alone or together for about 5.

Results from an additional 1. The risk of developing prostate cancer was also slightly increased in subjects taking vitamin E plus selenium or selenium alone, but the differences were not statistically significant. No differences were found among groups in the incidence of lung or colorectal cancers or all cancers combined.

Study staff members will continue to monitor participants’ health for up to 5 more years. However, prospective cohort studies of 87, women in the Nurses’ Health Study and 47, men in the Health Professionals Follow-up Study failed to replicate these results [ 34 ]. The American Cancer Society conducted an epidemiologic study examining the association between use of vitamin C and vitamin E supplements and bladder cancer mortality.

Of the almost one million adults followed between and , adults who took supplemental vitamin E for 10 years or longer had a reduced risk of death from bladder cancer [ 36 ]; vitamin C supplementation provided no protection. Evidence to date is insufficient to support taking vitamin E to prevent cancer.

In fact, daily use of large-dose vitamin E supplements IU of synthetic vitamin E [ mg] may increase the risk of prostate cancer. Age-related macular degeneration AMD and cataracts are among the most common causes of significant vision loss in older people.

Their etiologies are usually unknown, but the cumulative effects of oxidative stress have been postulated to play a role. If so, nutrients with antioxidant functions, such as vitamin E, could be used to prevent or treat these conditions.

Prospective cohort studies have found that people with relatively high dietary intakes of vitamin E e. A follow-up AREDS2 study confirmed the value of this and similar supplement formulations in reducing the progression of AMD over a median follow-up period of 5 years” [ 42 ]. Several observational studies have revealed a potential relationship between vitamin E supplements and the risk of cataract formation.

One prospective cohort study found that lens clarity was superior in participants who took vitamin E supplements and those with higher blood levels of the vitamin [ 43 ]. In another study, long-term use of vitamin E supplements was associated with slower progression of age-related lens opacification [ 44 ]. He began acting professionally in the early s and rose to prominence in the late s for his leading role as Troy Bolton in the High School Musical trilogy — During this time, he also starred in the musical film Hairspray and the comedy film 17 Again Efron has said that he would “flip out” if he got a “B” and not an “A” in school, as well as that he was a class clown.

Efron graduated from Arroyo Grande High School in [16] and was then accepted into the University of Southern California but did not enroll. He also attended Pacific Conservatory of the Performing Arts , a theatre company operating out of Allan Hancock College , a community college located in Santa Maria, California , where he performed in and Efron began acting in the early s with guest roles on several television series including Firefly , [18] ER , and The Guardian.

In , he began appearing as a recurring character in the first season of the WB series Summerland. He also appeared in some films, including the Lifetime television film Miracle Run , for which he earned a Young Artist Award nomination for his performance as one of two autistic twins. Efron’s career reached a turning point with the teen musical television film High School Musical , which premiered on the Disney Channel in January The film, which has been described as a modern adaptation of Romeo and Juliet , saw Efron playing the male lead Troy Bolton, a high school basketball player who feels conflicted when he finds himself interested in participating in the school musical with Gabriella Montez Vanessa Hudgens , a girl from the scholastic decathlon team.

The film, which also starred Ashley Tisdale , Lucas Grabeel , Corbin Bleu , and Monique Coleman in pivotal roles, became a major success and helped Efron gain recognition among teenage audiences. The film’s soundtrack was certified quadruple platinum by the RIAA , [20] making it one of the best-selling albums of the year in the United States.

Efron’s singing talents were disputed when it was revealed that his voice had been blended with Drew Seeley ‘s on the soundtrack, but Efron clarified that the songs written for a tenor were written before he was cast and therefore did not suit his more baritone vocal range. In his subsequent musical films, Efron did his own singing. Efron was next seen playing the role of Link Larkin in the musical comedy film Hairspray , based on the Broadway musical of the same name.

The film became a major commercial and critical success upon its release in July The film became a major blockbuster at the box office, and received mixed to positive reviews from critics.

He followed this with the commercially successful comedy 17 Again about a year-old man Matthew Perry who is transformed into his year-old self Efron after a chance accident. Efron’s next release was Richard Linklater ‘s period drama Me and Orson Welles , which premiered at the Toronto International Film Festival in September and received a wide release in late The film earned mostly positive reviews from critics.

He next played the title role in the supernatural romantic drama Charlie St. Cloud , which became a moderate success at the box office despite receiving mostly negative reviews from critics. Efron next appeared as a part of the large ensemble cast in Garry Marshall ‘s New Year’s Eve , which depicted a series of holiday vignettes of different groups of characters.

The film received almost unanimously negative reviews from critics, but became a major success at the box office. He also played a supporting role in the critically successful Liberal Arts , which premiered at the Sundance Film Festival in January and received a limited release later that year.

The film received mixed to negative reviews from critics. After lending his voice to the commercially successful computer-animated film The Lorax , he appeared as the male lead in the romantic drama The Lucky One , based on the novel of the same name by Nicholas Sparks.

The film became a major box office success despite negative reviews from critics. Both of the films received mixed reviews from critics. Efron’s first release of was the romantic comedy That Awkward Moment , on which he was also an executive producer. Jordan as three bachelors in New York City, became a moderate commercial success despite receiving mostly negative reviews from critics. Later in , Efron was seen in the adult comedy Neighbors , co-starring Seth Rogen.

The film revolved around a young couple, played by Rogen and Rose Byrne , who struggle to raise their baby daughter while living next to the house of a wild fraternity led by its president, played by Efron. The film became a major commercial success and earned mostly positive reviews from critics, who also added that Efron had successfully shed his “Disney pretty boy” image. Efron’s only release in was the moderately successful We Are Your Friends , in which he played a struggling DJ.

In January , he starred alongside Robert De Niro in the adult comedy Dirty Grandpa , about a straitlaced young man who begrudgingly indulges his grandfather’s unhinged personality by taking him on a vacation to Florida. The film received mostly negative reviews from critics for its crude humor, but became a commercial success upon its release.

The film followed the same couple Rogen and Byrne who team up with their former rival Efron to take down a hard-partying sorority led by a freshman Moretz.

In , Efron starred in Baywatch , an action comedy film version of the television series of the same name , released in May. Also in , Efron had a supporting role in two biographical films released in December, The Disaster Artist , a comedy-drama directed by and starring James Franco , and, as Phillip, in the musical The Greatest Showman , opposite Hugh Jackman.

People magazine reported in that Efron and Vanessa Hudgens began dating in during the filming of High School Musical , [33] [34] [35] although Us magazine reported, after the two broke up in December , that they “met in while making the first High School Musical flick, and became a romantic item about two years later. The couple split in April Efron sought treatment in early after struggling with alcoholism and substance abuse. He has been sober since June Law enforcement officials did not make any arrests because they viewed it as mutual combat.

In , Efron started his own production company under Warner Bros. Efron announced the creation of his YouTube channel in March Fans accused the platform of promoting a mainstream celebrity’s account, which they feared would overshadow lesser known creators and would make it more difficult for them to “break through. Efron turned to transcendental meditation TM to help decompress after struggling to separate himself from his role as serial killer Ted Bundy in his movie.

I did TM on the way home. Not while driving, but when you’re shooting a movie, one thing we can afford is a ride home, so on the way home I just do some TM and try to phase out of it. In December , Efron contracted a serious and potentially life-threatening illness, a “form of typhoid or similar bacterial infection “, while filming his adventure series Killing Zac Efron in Papua New Guinea.

He recovered with little issue. He moved to Australia [54] in early , [55] after selling his Los Angeles mansion and purchasing a property in Byron Bay, [56] basing himself there while doing a range of Australian-based projects.

From Wikipedia, the free encyclopedia. American actor and singer born It has been suggested that this article should be split into multiple articles. San Luis Obispo, California , U. See also: High School Musical cast discography. Retrieved October 16, Zac Efron. The Rosen Publishing Group. ISBN Retrieved March 11, Cal Coast News. July 29, Retrieved June 26, January 18, Retrieved August 24, The Jewish Exponent.

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